Skip to main content
What we do
Publications
NAD metabolism drives astrocyte proinflammatory reprogramming in central nervous system autoimmunity
Astrocyte immunometabolic regulation of the tumour microenvironment drives glioblastoma pathogenicity.
Control of tumor-associated macrophages and T cells in glioblastoma via AHR and CD39.
Prophylactic TLR9 stimulation reduces brain metastasis through microglia activation.
Cell composition analysis of bulk genomics using single-cell data.
Acute microglia ablation induces neurodegeneration in the somatosensory system.
Dissection of influenza infection in vivo by single-cell RNA sequencing
RIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS.
IL-10 dependent Tr1 cells attenuate astrocyte activation and ameliorate chronic CNS inflammation.
Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and central nervous system inflammation via the aryl hydrocarbon receptor.
http://www.nature.com/nm/journal/v22/n6/full/nm.4117.html
Regulation of astrocyte activation by glycolipids drives chronic CNS inflammation.
IL-27 acts on DCs to suppress the T cell response and autoimmunity by inducing expression of the immunoregulatory molecule CD39.
Our Research
Who we are
Our Team
Team
Former Lab Members
Gallery
Lab dinner. Nov 3, 2021
Bowling Dec 2018
Lab Cleaning and a day trip March 2019
Day Trip Nov 2019
Day trip Apr 2018
Our new MALDI
Group photos
Life the lab
Parents day 2018
Nov 2018
Join Us
News & updates
Lab News
Press (English)
Videos, interviews, & podcasts
World press (multilingual)
Contact
Give
Home
»
What we do
»
Publications
»
Astrocyte immunometabolic regulation of the tumour microenvironment drives glioblastoma pathogenicity.
Astrocyte immunometabolic regulation of the tumour microenvironment drives glioblastoma pathogenicity.
https://academic.oup.com/brain/article/145/9/2952/6670903
Nov 02, 2022
Show more...